Melting point	-70 ºC
Water Solubility	0.0503 g/L (20 ºC)
Boiling point	166-168 ºC
Flash point	60 ºC
Density	0.882
Refractive index	1.432-1.436

Symbol(GHS)	Xi
Hazard Codes	R36/37/38
SDS/MSDS	S26;S37/39

Source	SDS/MSDS Samples
没有数据	没有数据

Synthetic Route

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Name	CAS

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Articles

Peer-Reviewed Papers

Imaging Abeta plaques in living transgenic mice with multiphoton microscopy and methoxy-X04, a systemically administered Congo red derivative.William E Klunk et al.Journal of neuropathology and experimental neurology, 61(9), 797-805 (2002-09-17) Inhibition of huntingtin fibrillogenesis by specific antibodies and small molecules: implications for Huntington's disease therapy.V Heiser et al.Proceedings of the National Academy of Sciences of the United States of America, 97(12), 6739-6744 (2000-06-01) Amyloid binding ligands as Alzheimer's disease therapies.Virginia M-Y Lee Neurobiology of aging, 23(6), 1039-1042 (2002-12-10) Chrysamine G and its derivative reduce amyloid beta-induced neurotoxicity in mice.Kazuhiro Ishii et al.Neuroscience letters, 333(1), 5-8 (2002-10-29) Curcumin inhibits formation of amyloid beta oligomers and fibrils, binds plaques, and reduces amyloid in vivo.Fusheng Yang et al.The Journal of biological chemistry, 280(7), 5892-5901 (2004-12-14) Inhibition of polyglutamine aggregation in R6/2 HD brain slices-complex dose-response profiles.D L Smith et al.Neurobiology of disease, 8(6), 1017-1026 (2001-12-14) 99mTc-MAMA-chrysamine G, a probe for beta-amyloid protein of Alzheimer's disease.N A Dezutter et al.European journal of nuclear medicine, 26(11), 1392-1399 (1999-11-07) Chrysamine-G binding to Alzheimer and control brain: autopsy study of a new amyloid probe.W E Klunk et al.Neurobiology of aging, 16(4), 541-548 (1995-07-01) Development of small molecule probes for the beta-amyloid protein of Alzheimer's disease.W E Klunk et al.Neurobiology of aging, 15(6), 691-698 (1994-11-01) Chrysamine-G, a lipophilic analogue of Congo red, inhibits A beta-induced toxicity in PC12 cells.W E Klunk et al.Life sciences, 63(20), 1807-1814 (1998-11-20) In vitro drug treatments reduce the deleterious effects of aggregates containing polyAla expanded PHOX2B proteins.Eleonora Di Zanni et al.Neurobiology of disease, 45(1), 508-518 (2011-10-04)

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